Raw Vitamin E Rojmuaj nyob rau hauv siab concentration ntawm fat-soluble vitamins. Nws tswj hwm redox tshuav nyiaj li cas hauv lub cev thiab muaj thoob plaws hauv lub cev, suav nrog cov cell membranes thiab lipoproteins. Kab lus no qhia txog cov teebmeem antioxidant ntawm vitamin E.
Puas yog vitamin E muaj zog Antioxidant?
Cov kab mob aerobic muaj ob qho tib si enzymatic thiab tsis-enzymatic antioxidants. Enzymatic antioxidants muaj xws li catalase (CAT), glutathione
Glypeptide peroxidase (GPX) thiab superoxide dismutase (SOD). Non-enzymatic antioxidants muaj xws li albumin, anthocyanins, bilirubin, flavonols (xws li kaempferol, quercetin, thiab myricetin), isoflavones (xws li daidzin thiab genistein), flavonoids (xws li.
Verisitol, Hesperetin, thiab Naringenin), Flavanols (xws li Catechin thiab Gallocatechin), Folic Acid, Gallic Acid, Glutathione (GSH), Resveratrol, Ubiquitinol, Uric acid, phenolic acid, vitamin A (xws li astaxanthin, alpha-carotene, -carotene, beta-cryptoxanthin, lutein, lycopene), vitamin C (xws li ascorbic acid), vitamin E, vitamin K Tos. Piv rau exogenous fat-soluble vitamins nyob rau hauv lub cev, vitamin E yog ubiquitous nyob rau hauv cell membranes thiab yog tam sim no nyob rau hauv lub siab tshaj plaws concentrations thoob plaws hauv lub cev. Yog li, vitamin E ua lub luag haujlwm tseem ceeb hauv kev tswj hwm kev sib cuam tshuam redox hauv vivo.
VE Antioxidant ua haujlwm li cas?
Cov nyhuv antioxidant ntawm vitamin E feem ntau yog inhibit lipid peroxidation. Qhov no yog vim vitamin E muaj nyob rau ntawm lub cell membrane thiab koom nrog lipid peroxidation cov tshuaj tiv thaiv ntawm cov cell membrane. Txij li thaum ROS hnov mob thiab ploj mus sai heev hauv vivo, nws nyuaj rau ncaj qha rau ROS. Nws yog tam sim no ntseeg hais tias lub mechanism ntawm kev txiav txim yog inhibit qhov lipid peroxidation saw cov tshuaj tiv thaiv downstream ntawm ROS-induced oxidative kev nyuaj siab txoj kev. Lipid peroxidation ua tiav peb txheej txheem: "pib", "amplification" thiab "termination".